Memo re: Lloyd, Carl and Ray ADEN peptic ulcer disease (Attachment in Separate Entry)
Email From Gary Aden to Gary Aden
Email Dated 3/13/2001
The article in the Sept. 20, 1993 New Yorker describes the work of
Dr. Barry Marshall reported in the Annals of Internal Medicine wherein
the cause of peptic ulcer disease is attributed to an infectious disease
bacterium previously thought to be only an oppotunistic pathogen of
relatively little signficance in contrast to previous conventional
wisdom which theorized that the cause was a combination of genetic,
psychophysiologic, and toxic food/alcolhol/nicotine interaction with the
individual's duodenum. Additionally, the article develops evidence of a
link between gastic cancer in the mother of the individual and
subsequent susceptibility to the bacterium.
In the case of the above individuals, it is known that both Lloyd
and Carl suffered from duodenal ulcer disease and Ray may have had the
same disease. All were somewhat chronically restless and nervous
individuals, especially Lloyd and Carl. All smoked and drank to excess.
In Carl's case, the illness was often precipitated by separations from
signficant individuals in his life(this characteristic of the illness in
fact became a source of family jokes). Carl's near lifetime suffering
came to an end with successful surgery. The cause of the mother's death
was not legible on the available copy of the death certificate although
the word carcinomatosis could be discerned. It is known that she had near
death encounter with a bout of typhoid fever.
This memo brings fresh perspective to te notion of any genetic
predispostion to duodenal ulcer in the descendants of John W. Aden and
Jessie Mechling Aden.
Gary C. Aden, MD
Email Dated 3/13/2001
The article in the Sept. 20, 1993 New Yorker describes the work of
Dr. Barry Marshall reported in the Annals of Internal Medicine wherein
the cause of peptic ulcer disease is attributed to an infectious disease
bacterium previously thought to be only an oppotunistic pathogen of
relatively little signficance in contrast to previous conventional
wisdom which theorized that the cause was a combination of genetic,
psychophysiologic, and toxic food/alcolhol/nicotine interaction with the
individual's duodenum. Additionally, the article develops evidence of a
link between gastic cancer in the mother of the individual and
subsequent susceptibility to the bacterium.
In the case of the above individuals, it is known that both Lloyd
and Carl suffered from duodenal ulcer disease and Ray may have had the
same disease. All were somewhat chronically restless and nervous
individuals, especially Lloyd and Carl. All smoked and drank to excess.
In Carl's case, the illness was often precipitated by separations from
signficant individuals in his life(this characteristic of the illness in
fact became a source of family jokes). Carl's near lifetime suffering
came to an end with successful surgery. The cause of the mother's death
was not legible on the available copy of the death certificate although
the word carcinomatosis could be discerned. It is known that she had near
death encounter with a bout of typhoid fever.
This memo brings fresh perspective to te notion of any genetic
predispostion to duodenal ulcer in the descendants of John W. Aden and
Jessie Mechling Aden.
Gary C. Aden, MD
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